Sustanon 250 is a mixture of 4 short-acting testosterone and long-acting testosterone. It is a male hormone，t will be converted into estrogen and will also cause the body to store water. It has no liver toxicity. It can quickly increase muscle circumference and improve strength. It is one of the most basic steroids. It is suitable for beginners and is also suitable for use in the muscle gain or fat loss stage.
Product Performance
Strength increase:
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Muscle gain:
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Fat/water reduction:
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Side effects:
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Effect retention:
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Packaging: oil
Specification: 250mg/10ml
Appearance: injection
Dosage: 1ml~3ml/week
Cycle: 10~12 weeks
Sustanon 250 is a 250mg oil-based testosterone injection containing 4 types of testosterone with different ester groups: testosterone propionate (30mg), testosterone phenylpropionate (60mg), testosterone isocaproate (60mg) and testosterone decanoate (100mg). Mixed testosterone is a testosterone that is specially designed to produce a rapid sustained release effect. It is usually injected every 3-4 weeks in clinical practice. It is a modification of standard testosterone such as testosterone propionate and enanthate, so the duration of the drug effect is shortened. Like other testosterone products, Sustanon 250 is a highly potent anabolic steroid with significant androgenic properties. Sustanon 250 is most commonly used during the bulking phase to provide an immediate increase in muscle strength and mass.
Sustanon 250 is probably the most popular injectable steroid for athletes. However, it must be emphasized that this is not because of the special binding potency of testosterone (esters only affect the release of testosterone), but because people feel that it is better and more valuable to have four different testosterones working together. In most cases, you will feel that you are getting more value for your money.
The testosterone propionate in the ingredients is more painful when injected. Because the carbon chain of the propionate ester is very short, it will irritate the tissue at the injection site. Many people with sensitive constitutions deliberately avoid using this steroid because of the obvious pain and low-grade fever after injection, which may last for several days.
Directions for use
For the treatment of androgen deficiency, the prescription guidelines call for 250 mg injections every 3 weeks. Although Sustanon 250 is active for a long time in the body, it still needs to be injected every 7-10 days to ensure muscle gain. If you are an athlete, you can increase the dose and the hormone will reach a more stable level. The dosage range for male athletes is between 250-270 mg for 6-12 weeks. This dose is enough for most people to feel their increasing muscle size and strength.
Sustanon 250 is usually used in the muscle bulking training phase. It has almost no water retention reaction, so it is suitable for users who focus more on hard muscle strength than muscle lines. Sustanon 250 can also be used in the muscle building phase. It can be injected at a low dose (125-250 mg every 7-10 days) or used with aromatase inhibitors to control hormone levels. It is a very effective anabolic drug used alone, but it works better with other anabolic/androgenic steroids, such as 200-400 mg/week of Boldenone, Primobolan, and long-acting Durobolin, which have been proven to be non-hepatotoxic by a large number of results. Testosterone has a wide range of uses and can be combined with other anabolic/androgenic steroids as needed to achieve different effects.
Although it is generally not recommended to use excessive doses of the drug, some athletes still inject 1000 mg or more per week. If it exceeds 750 mg/week, water retention may occur, which may lead to weight gain and weaken the muscle-building effect. Practice has shown that “super high doses” are ineffective (not to mention potentially dangerous).
Sustanon 250 Side Effects (Estrogen):
Testosterone is easily aromatized by the body to estradiol (estrogen). Aromatase (estrogen synthase) is responsible for testosterone metabolism. Therefore, elevated estrogen levels may cause side effects such as water retention, increased body fat, and gynecomastia. Testosterone is considered a relatively mild estrogenic steroid. To prevent estrogenic side effects, it is recommended to take an anti-estrogen product such as clomiphene or tamoxifen. It is also possible to alternate with an aromatase inhibitor such as anastrozole to control estrogen synthesis. However, aromatase inhibitors are much more expensive than anti-estrogen products and may have a negative impact on blood lipids.
Estrogenic side effects usually occur when used in high doses (above normal therapeutic levels) and depend on the method of use. Therefore, when taking high doses, an anti-estrogen product or aromatase inhibitor should be taken at the same time. Because high doses of testosterone may cause water retention and loose muscle definition, this drug is generally considered a poor choice during dieting/training phases. However, its mild estrogenic properties make it ideal for the muscle-building phase, where water retention increases muscle strength and size and helps the body create a strong metabolic environment.
Side Effects (Androgen):
Testosterone is the primary androgen responsible for maintaining male secondary sexual characteristics. Possible side effects of elevated testosterone levels include oily skin, acne, body hair growth, and may exacerbate hereditary hair loss (androgenic alopecia). Those concerned about hair loss may choose a relatively mild steroid, nandrolone decanoate. Women should be aware that when using stronger androgens such as testosterone, masculinizing side effects may occur, including: deepening of the voice, irregular menstruation, skin wrinkles, facial hair growth, clitoral hypertrophy, etc.
Testosterone levels are relatively higher in target tissues that are more sensitive to androgens, such as the skin, scalp, and prostate, because they reduce dependence on dihydrotestosterone (DHT). 5-alpha reductase is responsible for metabolizing testosterone. Concomitant use of 5-alpha reductase inhibitors such as finasteride or dutasteride interferes with the site-specificity of testosterone action, reducing the likelihood of androgenic side effects with testosterone medications. Note that both anabolic and androgenic effects are mediated through androgen receptors. It is not possible to completely separate the anabolic and androgenic properties of testosterone, even if all 5-alpha reductases are inhibited.
Side Effects (Hepatotoxicity):
Testosterone has no hepatotoxic effects and is unlikely to produce hepatotoxic side effects. One study examined the hepatotoxic potential of high-dose testosterone by administering 400 mg of the hormone daily (2800 mg per week) to a group of male subjects. The results showed that oral steroids were more likely to reach higher peak concentrations in liver tissue than intramuscular injections. Subjects were given daily steroids for 20 days, and there were no significant changes in liver enzyme values, including serum albumin, bilirubin, alanine aminotransferase, and alkaline phosphatase.
Side Effects (Cardiovascular):
Sustanon 250, as an anabolic/androgenic steroid, may have a deleterious effect on serum cholesterol. This includes lowering HDL (good) cholesterol and increasing LDL (bad) cholesterol, which may shift HDL toward LDL and increase the risk of atherosclerosis. The effects of anabolic/androgenic steroids on lipid profiles vary depending on the dose, route of administration (oral or injection), type of steroid (aromatized or non-aromatized), and level of resistance to hepatic metabolism. With its resistance to hepatic metabolism and oral route of administration, it has a significant effect on cholesterol levels in the liver. Anabolic/androgenic steroids may also affect blood pressure and triglycerides, reduce subcutaneous relaxation, thicken the left ventricle, and increase the potential risk of cardiovascular disease and myocardial infarction.
Testosterone has a much smaller effect on cardiovascular risk than anabolic steroids. This is partly due to its resistance to hepatic metabolism, which makes the liver less resistant to cholesterol control. Aromatization of testosterone to produce estradiol also helps to mitigate the negative effects of androgens on lipid profiles. One study showed that taking 280 mg of testosterone ester (heptanoate) per week did not significantly affect HDL (high-density lipoprotein) cholesterol after 12 weeks, but when switching to aromatase inhibitors, HDL cholesterol values were significantly reduced (25%). If 300 mg of testosterone ester (heptanoate) was taken per week, HDL cholesterol values were only reduced by 13% after 20 weeks, and when the dosage was increased to 600 mg, it was reduced by 21%. Therefore, if aromatase inhibitors are added to testosterone therapy, their negative effects must be considered.
Because estrogen has a positive effect on blood lipids, taking aromatase inhibitors such as tamoxifen or clomiphene is more beneficial to cardiovascular health. These drugs play a partial estrogen role in liver metabolism, helping to improve blood lipid profiles and offset some of the negative effects of androgens. If 600 mg or less of testosterone is taken per week, the effect on blood lipids is more obvious but not so exaggerated, so there is no need to take anti-estrogen. This dose also did not significantly alter LDL/VLDL/cholesterol, triglycerides, apolipoprotein B/C, C-reactive protein, and insulin sensitivity, important risk factors for cardiovascular disease. Moderate doses are generally considered safest.
To help reduce cardiovascular stress, it is recommended to maintain an active cardiovascular exercise program and minimize saturated fat, cholesterol, and simple carbohydrate intake during active AAS administration. Supplementation with fish oil (4 grams per day) and a natural cholesterol/antioxidant formula such as Lipid Stabil or a product with similar ingredients is recommended.
Side Effects (Testosterone Suppression):
Adequate amounts of anabolic/androgenic steroids can effectively promote muscle growth and suppress endogenous testosterone secretion. Testosterone is the primary androgen and has a strong negative feedback on endogenous testosterone secretion. Testosterone drugs also have a significant effect on the hypothalamus’ regulation of natural steroid hormones. Without the intervention of testosterone-stimulating substances, testosterone levels generally return to normal 1-4 months after stopping medication. Note that long-term drug abuse may lead to hypogonadism and require medical intervention.